Little known study reveals that glyphosphate induces human breast cancer

A study published in the Journal of Toxicology on the 10th of June 2013 categorically links the induction of human to glyphosphates via estrogen receptors.

The study authors focused their research on comparing the prevalence of cancer when the endocrine system is disrupted and they found that glyphosphate had a significant impact in -dependent breast cancer, T47D cells, but not in hormone-independent breast cancer, MDA-MB231 cells. It was determined that the estrogen element significantly induced the response to Glyphosphate and acted as a mediation conductor.

The results of this study conclusively demonstrated that low and environmentally relevant concentrations of glyphosate possessed estrogenic activity. Glyphosate-based herbicides are widely used for , and the results also found that there was an additive estrogenic effect between glyphosate and genistein, a phytoestrogen in soybeans.

One of the most prolific and controversial studies involving Roundup, (a Glyphosphate herbicide), and Monsanto’s genetically engineered GMO corn, (strain NK603), involved the Séralini 2012 study published in the journal of Food and . The study produced results that were based on a long term feed study and demonstrated a wide variety of tumor development.

The most recent published study underlines the associated with Glyphosphate and their release into the environment as herbicides or contained in crop genetically engineered to be resistant to glyphosphate.

Source

Toxicol. 2013 Jun 10. pii: S0278-6915(13)00363-3. doi: 10.1016/j.fct.2013.05.057. [Epub ahead of print]
Glyphosate induces human growth via estrogen receptors.Thongprakaisang S, Thiantanawat A, Rangkadilok N, Suriyo T, Satayavivad J.

Gilles-Eric Séralini, Emilie Clair, Robin Mesnage, Gress, Nicolas , Manuela , Didier Hennequin, Joël Spiroux de Vendômois, “Long term toxicity of a Roundup herbicide and a Roundup-tolerant genetically modified maize,” Food and , Available online September 19, 2012.

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