New Study comfirms that BT toxin appears in the blood.

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Another study has confirmed the mountain of evidence that substantiates the toxicity of BT protein in mammals and the prevalence in the .

Monsanto has used the gene from (Bt) to genetically modify certain crops to be Bt resistant. produces BT-toxin. It’s a pesticide; and it breaks open the stomach of certain insects and kills them.

The new study reveals that the Cry1Aa, Cry1Ab, Cry1Ac or Cry2A have toxic effects in the blood of mice and that the claims by policy regulators of the presumed nontoxicity of Bt toxin to mammals, on which all of are based, is false.

The mechanism of operation of BT toxin on insects (the target population for the pesticide) is to break holes in the gut and rupture cells. In mice in this experiment , caused to rupture and is highly disconcerting as BT toxin has crossed the placenta and was found in the umbilical cord in over eighty percent of mothers tested suggesting that it crosses into the of babies.
http://foodexposed.co.za.www100.jnb2.host-h.net/gm-study-on-maternal-exposure-to-gm-pesticides-receives-little-exposure/

The study authors stated: “It has been reported that Cry toxins exert their toxicity when activated at of the digestive tract of susceptible larvae, and, because the physiology of the does not allow their activation, and no known specific receptors in mammalian have been reported, the toxicity [of] these MCAs [] to mammals would negligible. However, our study demonstrated that Bt spore-crystals genetically modified to express individually Cry1Aa, Cry1Ab, Cry1Ac or Cry2A induced hematotoxicity, particularly to the erythroid lineage. This finding corroborates literature that demonstrated that -solubilized Bt spore-crystals caused in vitro hemolysis in cell lines of rat, mouse, sheep, horse, and human erythrocytes and suggested that the plasma membrane of susceptible cells (erythrocytes, in this case) may be the primary target for these toxins”.
” In conclusion, results showed that the Bt spore-crystals genetically modified to express individually Cry1Aa, Cry1Ab, Cry1Ac or Cry2A can cause some hematological risks to vertebrates, increasing their toxic effects with long-term exposure. Taking into account the increased risk of human and animal exposures to significant levels of these toxins, especially through diet, our results suggest that further studies are required to clarify the mechanism involved in the hematotoxicity found in mice, and to establish the toxicological risks to non-target organisms, especially mammals, before concluding that these microbiological control agents are safe for mammals.”

The toxicity of Bt proteins in mammalian cells was also the subject of an in vitro (test-tube) study (Mesnage et al., 2012; http://onlinelibrary.wiley.com/doi/10.1002/jat.2712/abstract). In this study, Bt toxin Cry1Ac was found to be substantially toxic to human cells, raising substantial concerns in the human population.

Source

Mezzomo, B. P., et al. (2013). Hematotoxicity of as spore-crystal strains Cry1Aa, Cry1Ab, Cry1Ac or Cry2Aa in Swiss albino mice. J Hematol Thromb Dis 1(1). http://dx.doi.org/10.4172/jhtd.1000104

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